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Jul 20, 2011. Forskolin also activated adenylyl cyclase and enhanced cyclic. AMP accumulation. which appears to be a pivotal, but an indirect mediator of pro-. Life. Sci 33: 1401–1408. Tanaka M, Watanabe Y, Yoshimoto K (2009) Regulation of relaxin 3 gene expression via cAMP-PKA in a neuroblastoma cell line.
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Jun 7, 2016. A recent study has identified an essential role for the dual zipper-bearing kinase DLK in the pro-regenerative effect of a conditioning lesion in adult DRG. the catalytic subunit (PKACA) (Li et al., 1995) led to an enhanced growth response of Drosophila motoneuron axons after nerve crush injury (Figure 1A).
Jun 13, 2017. p53-mediated transcription of the pro-opiomelanocortin. (POMC) gene (Cui et al., 2007). Proteolytic. cAMP agonist forskolin was shown to rescue the cAMP-MITF – eumelanin pathway in Mc1r-deficient mice. The half-life of melanin in skin is thought to be several weeks and diminishes primarily after.
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Forskolin significantly enhanced heregulin-stimulated expression of cyclin D and phosphorylation of the retinoblastoma gene product. The cells were plated in 24-well culture dishes and transfected with 0.2 μg of cyclin D reporter plasmid for 3 h by using Lipofectamine-Plus in serum-free OptiMEM (Life Technologies, Inc.).
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Nerve Growth Factor-Induced Differentiation of. – Karger Publishers – Apr 14, 2010. adenylyl cyclase-activating property of forskolin in PC12 cells. This effect. factor (s) were responsible for the enhanced responses to. Life Sci 2007; 81: 267– 271. 48 Tan CM, Kelvin DJ, Litchfield DW, Ferguson. SSG, Feldman RD: Tyrosine kinase-mediat- ed serine phosphorylation of adenylyl cy- clase.
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However, PGE2, forskolin, and PMA enhanced the stability of IL-8. mRNA transcripts. Furthermore, dexamethasone inhibited PGE2-, forskolin-, and dibutyryl cAMP-induced, but not PMA-induced, IL-8 protein production. These. Total RNAs were isolated from T84 cells with TRIzol reagent (Life Tech- nologies) , and.
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